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Inhaled cigarette smoke first encounters the airway epithelium, where it may induce oxidative stress by both acute effects of its reactive components and by the intracellular generation of endogenous reactive oxygen species (ROS) by mitochondria [16]

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Archived from the original on December 11, 2011

Emerging studies suggest that CS-induced disruption of respiratory microbial communities may contribute to the pathogenesis of other respiratory diseases, including laryngitis, asthma and acute respiratory distress syndrome (ARDS)