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Also, the upregulation inflammasome-related genes (CXCL1, CXCL2, NOD2, and ASC) in smoking and vaping have been observed, revealing the negative effect of vaping/smoking on the inflammation and susceptibility to SARS-CoV-2 infection [328]
found that increased biofilm formation of Streptococcus pneumoniae and inactivation of pneumolysin induced by exposure to cigarette smoke condensate are likely to favor microbial colonization and persistence, both being essential precursors of pneumococcal disease [35]
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