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found that endothelial cell dysfunction, which is characterized by low nitric oxide (NO) bioavailability, greater reactive oxygen species production, the activation of abscission enzymes, and impaired extracellular lysosome-associated organelle function, triggers the degradation of the EG (57), leading to shear stress defects

The application of L-asparaginase as a plasma Gln-depleting agent with a clear therapeutic effect in solid tumors warrants further investigation