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KEAP1/NRF2 mutant cells are protected against cysteine-starvation induced ferroptosis due to elevated GCLC expression and limited glutamate accumulation, and dual targeting of cystine import and GCLC activity can overcome the resistance of NRF2 active cells to ferroptosis (Kang et al., Cell Metab 2021)

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Both are investigated in metabolic research, but Cagrilintide focuses on satiety and feeding behavior, while Retatrutide targets broader metabolic regulation through incretin pathways

Kang et al