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Obesity-associated co-morbidities are complicatedly linked with an expansion in reactive oxygen species and consequent oxidative stress, which grounds a state of redox imbalance (Reference Savini, Catani and Evangelista2,Reference Asbaghi, Ghanavati and Ashtary-Larky3)

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An alternative explanation, which could potentially explain both sets of experiments, is that exported GSH is broken down into constituent amino acids in the blood that are rapidly reimported into the liver cells