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UH supervised execution of the clinical trial at Profil Institute as principal medical investigator
Following exposure of rats to 3.5 h of WRS, a significant decrease of SOD activity (by about 30%) was observed
In both pathways, iron overload led to an overall reduction in the expression levels (cardiac muscle contraction pathway Figure 7A and citrate cycle Figure 7B)
Mechanisms of ascorbic acid stimulation of norepinephrine synthesis in neuronal cells
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