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The intricate dance of signaling molecules, encompassing IL-6, TGF, IL-23, and IL-1, meticulously orchestrates the Th17 cell differentiation process

Pain hypersensitivity is the consequence of early posttranslational changes, both in the peripheral terminals of the nociceptor and in dorsal horn neurons, as well as later transcription-dependent changes in effector genes, again in primary sensory and dorsal horn neurons (4)