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GPx7 likes to interact with the domain of PDI, and intramolecular cooperation between the two redox-active sites of PDI increases the activity of the Ero1/GPx7/PDI triplet, thereby promoting protein folding mechanisms (Wang et al., 2014) (Figure 6)

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GHK-Cu works primarily at the structural level rebuilding extracellular matrix proteins and activating genetic programs for tissue renewal

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