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In this study, we identified a neural circuit in male mice that compensates for the reduction in the number of glutamatergic neurons in the BLA due to heroin dependence, extending from the NTS to the BLA
Interestingly, although DAF-16 is similarly activated in both glp-1(-) and fem-3(-) mutants, glp-1(-) shows a greater lifespan extension compared to fem-3(-) , again indicating that whereas DAF-16 is important, other factors are also likely to be key in modulating longevity in response to sterility 6,14,15,16,66
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Nikolova, V
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