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Accordingly, excess DPP-4, derived from adipocytes and/or hepatocytes, may act as a local mediator of inflammation and adipose/hepatic tissue insulin resistance, thereby forming one link between obesity and the pathogenesis of T2DM and metabolic disease (Figure 3)

Ancient atmospheres and the evolution of oxygen sensing via the hypoxia-inducible factor in metazoans

Cell Death Differ 31:9991012 Gyan E, Frisan E, Deschemin J-C et al (2006) Both the endoplasmic reticulum and the mitochondria are involved in apoptosis of erythroid precursors in low grade Myelodysplastic syndromes