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Notably, breast cancer cells achieve metabolic adaptation by hijacking the autophagy mechanism: under glucose-deprivation conditions, MANF-mediated mitophagy eliminates dysfunctional mitochondria through the PRKN/Parkin-dependent pathway, maintains the ATP supply and promotes tumour survival [112], suggesting that targeting the MANFPRKN interaction can break the drug resistance barrier of breast cancer under metabolic stress

Tirzepatide has also shown some promising results with up to 8% absolute reduction in liver fat content in a RCT

Abdelhamed Hamdy@Abdelhamed6261EFFECTS OF GLP-1 RECEPTOR AGONISTS ON HYDRATION AND ELECTROLYTE BALANCE6:56 PM Jun 27, 20262.1KViews1134223

BPC-157 acts as a master signaling molecule, with microarray analysis revealing a 7-fold up-regulation of Growth Hormone Receptor (GHR) in tendon fibroblasts by the third day of treatment