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This indicates that NCOA4 functions upstream of SFXN1 and implicates NCOA4-mediated autophagy and ferroptosis in the development of cardiac hypertrophy [159, 160]

This complex biochemical conversion occurs through two main interconnected pathways: the Tricarboxylic Acid (TCA) cycle, also known as the Krebs cycle, and the Electron Transport Chain (ETC)

It helps regenerate oxidized glutathione back into its active form, allowing the antioxidant to continue protecting cells from oxidative stress