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Early, small trials suggested a beneficial effect of ALCAR with respect to cognitive decline (98-100), whereas later, larger trials found little-to-no effect compared to placebo (101-103)

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found through experiments that compared to the TMAO group, synaptic structural damage significantly improved in the AOB group, characterized by regular synaptic morphology, enhanced vesicle distribution within the presynaptic region, and more obvious synaptic clefts

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