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As a thymosin beta-4 fragment it is studied for actin regulation and cell migration helping fibroblasts and other repair cells physically reach and organize at the site that GHK-Cu is signaling to rebuild

Because more free peptide reaches tissues, in vitro receptor-activation studies show that IGF-1 LR3 produces near-maximal IGF-1R phosphorylation at lower molar concentrations than the native ligand [2]

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[DOI] [PubMed] [Google Scholar] 72.Drucker DJ