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This indicates that tryptophan metabolite suppresses T-cell responses by inhibiting DCs activation ( Glutamate/glutamine Glutamine is the most abundant amino acid in the blood and the main precursor of glutamate ( 2 through the TCA cycle to provide energy to cells ( DCs expressed glutamate transporters, glutamate-specific vesicular transporters and were capable of fast glutamate release through a Ca 2+ -dependent mechanism ( 2+ channels, triggering of the sustained Ca 2+ signal, and core crushing of T cells to thymocyte-DC contact zones ( In addition to serving as a carbon source, glutamate is also an important nitrogen source for the synthesis of nucleotides, glucosamine, and other non-essential amino acids through transamination or deamination ( Given the impact of xCT on antigen-presenting potential of DCs, and the role of xCT in cancer exhaustively described by previous reports (including ferroptosis ( Oxidative stress changes APC function, Glutathione can interfere with the release of DCs cytokines and regulate Th1 differentiation

Field AE, Byers T, Hunter DJ, et al

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