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In parallel, metabolic interventions, including high-calorie diets and medium-chain triglyceride supplementation, are undergoing clinical evaluation to counteract the hypercatabolic state characteristic of ALS [79], a systemic reflection of the severe energetic crisis driven by motor neuron mitochondrial failure

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Moreover, increased pressure has been shown to enhance cancer cell migration and resistance to the anticancer drug doxorubicin by upregulating the water channel aquaporin 1 [52] and the efflux transporter ABCC1 [53], respectively