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Patients with glutathione synthetase deficiency accumulate the dipeptide -glutamylcysteine and cysteine in fibroblasts [24]

No published study has linked BPC-157 to tumorigenesis, and Sikiric's group has explicitly noted the absence of neoplastic findings in chronic dosing models 1

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The thiomethyl shunt involves the activity of CCBL to cleave cysteine S-conjugates, and subsequent methylation with an active form of methionine to produce thiomethyl metabolites ( 9, 21 ), meaning that APAP-SG and NAC-APAP were probably biotransformed to APAP-cysteine beforehand