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Observations of neurodevelopmental or neurodegenerative disease in humans, triggered by mutations in various proteins involved in autophagy, provide additional support for this hypothesis, including those affecting PINK1, Parkin, OPTN, TBK1, ATL1, SQSTM1, and WDR81, as well as ATG5, AP4, HTT, WIPI4, and DYNC1H1 (Zhu et al

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Reliable glutathione profiling helps researchers decide whether interventions reduce oxidative stress, whether bioprocesses are under control, or whether quality standards are being metturning raw data into actionable insights

doi: 10.2337/db14-0668 111 KrausC.CastrenE.KasperS.LanzenbergerR