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Finally, we emphasize the indispensable role of advanced human models, such as human pluripotent stem cell-derived islets

Therefore, our meta-analysiss conclusion was more conservative

Additionally, exendin-4 treatment mitigated high glucose-induced mitochondrial dysfunction, as evidenced by reduced expression levels of mitochondrial function-associated genes mitochondrial calcium uniporter (MCU) and uncoupling protein 3 (UCP3), as well as the mitochondrial fission-related gene dynamin-related protein 1 (DRP1) [79]

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