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Hyperglycaemia activates the NF-B and JNK pathways, leading to pro-inflammatory milleu, impaired insulin signalling and insulin resistance in several insulin-responsive tissues, such as hepatocytes, skeletal muscles, adipocytes and endothelial cells [17, 18]

However, during increased electron flux, superoxide production may reach a critical threshold, which may allow trafficking of superoxide anions through the mitochondrial transition pore into the cytosol, in which further damage and/or the activation of additional ROS sources may occur

Newer GLP-1 drugs in high demand cost more than older options in the same drug family

Skin reactions including injection site reactions and occasional hypersensitivity symptoms occurred but were predominantly mild