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Furthermore, chronic treatment with the GLP-1 mimetic liraglutide induces similar improvements on glucose tolerance despite the loss of PNS or CNS-GLP-1R expression, indicating that non-neuronal GLP-1R signaling is sufficient for the long-term improvements in glucose homeostasis induced by liraglutide in mice (Sisley et al., 2014)
Animal models showed exercise-equivalent effects (mitochondrial biogenesis, fat oxidation, endurance gains) without physical activity
The rate of protein synthesis in hematopoietic stem cells is limited partly by 4E-BPs
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