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Mutations in UBQLN2 have recently been shown to cause dominant X-linked inheritance of ALS/dementia 126

The consequence of this interaction is the breakdown of immune tolerance, manifested by the proliferation of follicular helper T (Tfh) and Th17 cells, impaired function of Tregs, aberrant B-cell activation leading to excessive autoantibody production, elevated levels of inflammatory mediators, multiorgan inflammation, and tissue damage, ultimately culminating in the development of autoimmune disorders

However, some individuals may experience mild digestive upset or allergic reactions