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(2024) showed that GHRP-6 prevented doxorubicin-induced myocardial and extra-myocardial damage in rats by attenuating pro-oxidant activity, enhancing antioxidant reserves, protecting mitochondrial ultrastructure, and upregulating the anti-apoptotic gene Bcl-2

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[10] [18] Preliminary information on side effects and safety was supplied by case studies on the use of NAD+ for the treatment of drug addiction

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